By Jo Nova

If anyone says “but this was only a few people” — the absolute scandal here is that every nation in the West should have been doing studies like this nearly two years ago and by the hundreds.

German pathologists studied heart muscle cells in 25 people who died at home within a month of getting vaccinated. Of these,  five people had strange strange clusters of white blood cells infiltrating their heart muscle and all of those died within just a week of being vaccinated. In three of those cases, the results were quite clear, and there were no other potential causes of myocarditis, and no obvious heart problems, and the link to the vaccination was quite strong. “Further studies are needed”.

Constantin Schwab and others searched back through historic records and were struck that during the last 20 years of autopsy service at Heidelberg University Hospital they had not seen any “comparable myocardial inflammatory infiltration.” It really was unusual.

In this case the myocarditis was not necessarily severe, but the mild localized inflammation in clusters may have interfered with the pattern of contraction of heart muscle, causing fibrillation — where contracting cells become unsynchronized. In that trembling state, the heart can’t pump blood and death is rapid unless someone gets “defibrillated” and the electric shock resets the pattern.

Dr John Campbell describes this study as a “smoking gun” in terms of the cause and effect connection with vaccines.

And in one of those uncanny coincidences, wouldn’t you know it, but communal defibrillators are being installed on the front of houses in a trial in one suburb of Melbourne this year. Because fibrillation can happen to anyone…  see below.

Jennifer Margulis and Joe Wang, Epoch Times

Medical pathologists from Heidelberg University Hospital in Heidelberg, Germany have published direct evidence showing how people found dead after mRNA vaccination died. As this team of six scientists explore in their study, these mRNA-vaccinated patients suffered from heart damage because their hearts were attacked by their own immune cells. This autoimmune attack on their own heart cells then leads to their damaged hearts beating so many times per second that, once the tachycardia unexpectedly started, they died in minutes.

Died of Ventricular Tachycardia or Fibrillation

Ventricular tachycardia is when the heart begins beating so fast that it doesn’t have time to refill with blood between beats, so it is not adequately pumping blood. The problem originates from the ventricles: the chambers that push the blood out of the heart to the rest of the body.

In these images of heart muscle cells, inflammatory white blood cells appear as small dark spots. In D, as we zoom out we can see they are clustering sites of inflammation. These dark cells are not supposed to be there.

Visibly Damaged Hearts

Macrophages are large cells that are part of our immune system. When the immune system is functioning properly, our bodies use macrophages to attack infectious agents and other foreign matter. Macrophages are a key part of the innate immune system, helping with normal tissue development as well as with repairing damaged tissue, according to researchers from Northwestern University.

But in the case of the people who died suddenly within a month of being vaccinated, the body’s own macrophages permeated their heart muscle, chewing up the muscle and causing spots that disrupted the heart rhythm. This macrophage invasion appeared to have literally short-circuited the heart’s conduction of the electrical impulses, causing the heart to beat irregularly.

The irregular heartbeats led to a negative feedback loop, making the heart race faster and faster as it tries to right itself. When that happens, the heart is effectively pumping no blood, and the victim dies within seconds or minutes unless there is a defibrillator nearby—to deliver an electrical shock to the heart to help it get back into rhythm—and someone knows to use it immediately.

Heart muscle is special, primed from the beginning to contract in regular cycles for life. Cardiac cells generate their own electrical impulses, and they send signals to the cells around them so they can synchronize their contractions. If one cells “falls out” of the pattern, presumably the others around it bring it back in. But if enough cells lose the beat, chaos spreads.

Not so coincidentally, defibrillators are being rolled out in a Melbourne suburb

In Resevoir, 28 defibrillators have been installed at the front of homes, available for anyone to use, and no more than 400m away. It’s handy for all those times a teenage jogger collapses with ventricular tachycardia.

According to the experts Sudden Cardiac Arrests can impact and affect almost anybody, don’t you know: “it’s not about age it’s not about gender, it’s simply a killer across the board”, says CEO of St John Ambulance Victoria, Gordon Botwright.

Just like when we were kids, yeah? Which house on your street had a defibrillator when you were growing up…

The researchers speculate that the lop-sided nature of the inflammatory clusters (foci) could be due to intravenous delivery:

Interestingly, we recorded inflammatory foci predominantly in the right heart, which may suggest a gradual blood-stream derived dilution effect and based on this finding it is at least tempting to speculate that inadvertent intravascular vaccine injection may be contributive.

If they are correct, tragically these deaths might have been avoided with aspirated intramuscular injections which make sure they are not going straight into the bloodstream. It only takes a little bit longer…

As part of the study, they ruled out all the other viruses and bacteria that are capable of causing myocarditis:

Regarding potential causative infectious agents, FFPE samples of all cases and fresh frozen myocardial samples of cases  were tested for viral and bacterial genomes by a diagnostic panel with (reverse transcription) PCR (Enteroviruses, parvovirus B19, human herpesvirus 6, Epstein–Barr virus, human cytomegalovirus, varicella zoster virus, herpes simplex virus type 1 and 2, human herpesvirus 7, adenoviruses, borrelia spp., Toxoplasma gondii)

In one of the five victims, Human Herpes virus 6 was found, which means they can’t rule that out as a cause in that victim. But none of the infectious agents above were found in the other samples, leaving vaccination as the most likely reason.

The researchers explain their reasons they believe the Covid vaccines caused this:

In general, a causal link between myocarditis and anti-SARS-CoV-2 vaccination is supported by several considerations:

(A) a close temporal relation to vaccination; all cases were found dead within one week after vaccination, (B) absence of any other significant pre-existing heart disease, especially ischaemic heart disease or cardiomyopathy, (C) negative testing for potential myocarditis-causing infectious agents, (D) presence of a peculiar CD4 predominant T-cell infiltrate, suggesting an immune mediated mechanism. The latter criterion is supported by demonstration of a phenotypically identical T-cell infiltrate at the deltoidal injection site in one of the cases. It has to be emphasized, that a comparable (epi-)myocardial infiltration was neither found in any of the other 20 autopsies performed on bodies found dead within 20 days following an anti-SARS-CoV-2 vaccination nor in the age- and sex-matched cohorts from three independent periods from our autopsy-files.

In a small amount of good news, at least they did not find the same effect in the bodies of people who died three weeks after vaccination. So this effect, at least, appears to wear off.That does not mean the vaccines don’t cause other harm in other ways three weeks later (or three months later). But it shows this effect resolves quickly (or is fatal quickly). Sigh.

REFERENCE

Constantin Schwab et al (2022) Autopsy-based histopathological characterization of myocarditis after anti-SARS-CoV-2-vaccination,  Clinical Research in Cardiology

Diagram of the heart: Wapcaplet

h/t David Maddison, William Astley, Leo G, Lance, Krishna Gans, Richard K